Among many concerned about the way in which Monsanto (other biosphere destroyers are also available) has played the same game with data on safety as the statin producers and pushers [our law makers are either stupid, venal, or deliberately misinformed] is Stephanie Seneff – start at http://people.csail.mit.edu/seneff/ first 5 latest papers.

This promises to be a much bigger health issue than statins since everyone is potentially chronically exposed.

It would seem that different people tolerate being overweight better.
http://www.ncl.ac.uk/magres/research/diabetes/reversal.htm

“A crucial point is that individuals have different levels of tolerance of fat within liver and pancreas. Only when a person has more fat than they can cope with does type 2 diabetes develop. In other words, once a person crosses their personal fat threshold, type 2 diabetes develops. Once they successfully lose weight and go below their personal fat threshold, diabetes will disappear.

Some people can tolerate a BMI of 40 or more without getting diabetes. Others cannot tolerate a BMI of 22 without diabetes appearing, as their bodies are set to function normally at a BMI of, say 19. This is especially so in people of South Asian ethnicity.”

Some interesting work being done at Newcastle.

Something else that I’ve read of late being possibly connected to bringing about diabetes concerns high electrical EMF fields. The EMF diabetes connection I read was observational in scope, so not proof. It caught my attention though due to my grandfather. I had a relatively thin grandfather that developed diabetes. He eventually passed away from complication due to the disease. According to my father he lived most of his life pretty much under a power line. He also complained of muscle pains, particularly in the back, which I guess is thought to be another possible problem for some with high EMF fields. Of course hard to say what brought about granddad’s conditions.

Going further out on a limb with this, but my grandmother that lived in the house near the power line later developed Alzheimer’s disease. Her mother, that lived on the same small farm, and close to the power line too, also developed Alzheimer’s. So far they have been the only two family members that developed this disease in the family. I only mention as I’ve read some write Alzheimer’s develops from a 3rd type of diabetes, one that effects the mind. Of course speculation on this, but has me curious.

VERY good point, and I’ve never seen it mentioned in any of the studies that look at weight and shift work.

Guess they did not control for vitamin D. But although sunshine is a key source, our skins ability to make vitamin D declines with age making D one of the few vitamins supplements that could be needed. Another few reasons for increasing D-deficiency today are:
1/ Too many animals we eat are reared inside out o sun these days, (on wrong foods!)
2/ A low fat diet avoids the fat in animals where the important winter- D used to be stored. 3/ People eating cholesterol lowering sterols reduce the vital cholesterol that is needed for conversion to Vitamin D…
4/ We usually don’t reduce weight in winter any more so the summer D stays in our fat….

Finally re LDL: Since only glycated LDL sticks in arteries it means that avoiding high blood sugar spikes from modern fast carbs (breakfst cereal and sandwiches sticks out a mile….), reduces daily glycation and preserves useful LDL for brain, heart, Vitamin D-conversion, and more that it was made for.
By tackling root causes a lot of today’s medication can become redundant and with every one usually goes unwanted side effects.

p.s. Love your ‘A Great Day at the Office’

Longer (good) sleep periods would allow low blood sugar for longer time and hence less insulin in proportion which also means longer cleansing time for blood vessel plack and hence improved heart health, In part from what I read about how blood sugar needs to go down below 5 to reduce arterial plaque at Dr William Davis site. Trackyourplaque.

Same would of course apply to the apparent sticky culprits, the sugar coated or glycated (small) LDL. Is it easy to measure these villains? Or is a sufficiently low HbA1c level ok to know that few of them are at large? And if yes, what HbA1c level is required and what maximum% carbs can be recommended to stay there?
Of course the maximum permissible carb % drops with increasing age, but say for an average 40-50 year old ?